uPAR enhances malignant potential of triple-negative breast cancer by directly interacting with uPA and IGF1R-Reference-Cited by-同舟云学术

uPAR enhances malignant potential of triple-negative breast cancer by directly interacting with uPA and IGF1R

Published:2016-08-08 Issue:1 Volume:16 Page:
ISSN:1471-2407
Container-title:BMC Cancer
language:en
Short-container-title:BMC Cancer

Author:

Huber Michaela C.,Mall Rebecca,Braselmann Herbert,Feuchtinger Annette,Molatore Sara,Lindner Katrin,Walch Axel,Gross Eva,Schmitt Manfred,Falkenberg Natalie,Aubele Michaela

Funder

Wilhelm Sander-Stiftung

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Link

http://link.springer.com/content/pdf/10.1186/s12885-016-2663-9.pdf

Reference50 articles.

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3. Singh P, Alex JM, Bast F. Insulin receptor (IR) and insulin-like growth factor receptor 1 (IGF-1R) signaling systems: novel treatment strategies for cancer. Med Oncol. 2014;31:1.

4. Davison Z, de Blacquiere GE, Westley BR, May FEB. Insulin-like growth factor-dependent proliferation and survival of triple-negative breast cancer cells: implications for therapy. Neoplasia. 2011;13:504–15.

5. Nielsen TO, Hsu FD, Jensen K, Cheang M, Karaca G, Hu ZY, et al. Immunohistochemical and clinical characterization of the basal-like subtype of invasive breast carcinoma. Clin Cancer Res. 2004;10:5367–74.

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