Ell3 functions as a critical decision maker at the crossroad between stem cell senescence and apoptosis-Reference-Cited by-同舟云学术

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Ell3 functions as a critical decision maker at the crossroad between stem cell senescence and apoptosis

Published:2019-01-17 Issue:1 Volume:10 Page:
ISSN:1757-6512
Container-title:Stem Cell Research & Therapy
language:en
Short-container-title:Stem Cell Res Ther

Author:

Lee Jae-Yong,Lee Soo-Hong,Kim Kwang-Soo,Park Keun-Hong,Park Kyung-Soon

Funder

Ministry of Education, Science and Technology

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Biochemistry, Genetics and Molecular Biology (miscellaneous),Molecular Medicine,Medicine (miscellaneous)

Link

http://link.springer.com/content/pdf/10.1186/s13287-019-1137-9.pdf

Reference33 articles.

1. Smith JR, Pereira-Smith OM. Replicative senescence: implications for in vivo aging and tumor suppression. Science. 1996;273(5271):63–7.

2. Kalungi S, Wabinga H, Bostad L. The RB (pRb2/p16) and p53 (p14/p53/p21) tumor-suppressor pathways in endemic Burkitt lymphoma. J Pediatr Hematol Oncol. 2011;33(2):e54–9.

3. Wang TJ, Huang MS, Hong CY, Tse V, Silverberg GD, Hsiao M. Comparisons of tumor suppressor p53, p21, and p16 gene therapy effects on glioblastoma tumorigenicity in situ. Biochem Biophys Res Commun. 2001;287(1):173–80.

4. Serrano M, Lin AW, McCurrach ME, Beach D, Lowe SW. Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a. Cell. 1997;88(5):593–602.

5. Olsen CL, Gardie B, Yaswen P, Stampfer MR. Raf-1-induced growth arrest in human mammary epithelial cells is p16-independent and is overcome in immortal cells during conversion. Oncogene. 2002;21(41):6328–39.

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