A single rapamycin dose protects against late-stage experimental cerebral malaria via modulation of host immunity, endothelial activation and parasite sequestration-Reference-Cited by-同舟云学术

A single rapamycin dose protects against late-stage experimental cerebral malaria via modulation of host immunity, endothelial activation and parasite sequestration

Published:2017-11-09 Issue:1 Volume:16 Page:
ISSN:1475-2875
Container-title:Malaria Journal
language:en
Short-container-title:Malar J

Author:

Mejia Pedro,Treviño-Villarreal J. Humberto,Reynolds Justin S.,De Niz Mariana,Thompson Andrew,Marti Matthias,Mitchell James R.

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Parasitology

Link

http://link.springer.com/content/pdf/10.1186/s12936-017-2092-5.pdf

Reference41 articles.

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3. Schofield L, Grau GE. Immunological processes in malaria pathogenesis. Nat Rev Immunol. 2005;5:722–35.

4. McQuillan JA, Mitchell AJ, Ho YF, Combes V, Ball HJ, Golenser J, et al. Coincident parasite and CD8 T cell sequestration is required for development of experimental cerebral malaria. Int J Parasitol. 2011;41:155–63.

5. Haque A, Best SE, Unosson K, Amante FH, de Labastida F, Anstey NM, et al. Granzyme B expression by CD8+ T cells is required for the development of experimental cerebral malaria. J Immunol. 2011;186:6148–56.

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