Favorable effect of bortezomib in dense deposit disease associated with monoclonal gammopathy: a case report
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
http://link.springer.com/content/pdf/10.1186/s12882-018-0905-6.pdf
Reference19 articles.
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2. Zipfel PF, Skerka C. Complement regulators and inhibitory proteins. Nat Rev Immunol. 2009;9:729–40.
3. West CD, McAdams AJ, MacConville JM, et al. Hypocomplementaemic and normocomplementaemic persistent (chronic) glomerulonephritis; clinical pathologic characteristics. J Pediatr. 1965;67:1089–112.
4. Bomback AS, Appel GB. Pathogenesis of the C3 glomerulopathies and reclassification of MPGN. Nat Rev Nephrol. 2012;8:634–42.
5. Pickering MC, D'Agati VD, Nester CM, et al. C3 glomerulopathy: consensus report. Kidney Int. 2013;84:1079–89.
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Bortezomib-containing regiment in treating glomerulopathy with fibronectin deposits combined with monoclonal gammopathy of undetermined significance: a case report and literature review;Annals of Translational Medicine;2022-03
2. Use of Bortezomib in the Treatment of C3 Glomerulonephritis Refractory to Eculizumab and Rituximab;Kidney International Reports;2020-06
3. Monoclonal immunoglobulin mediates complement activation in monoclonal gammopathy associated-C3 glomerulonephritis;BMC Nephrology;2019-12
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