A vital role for Angptl3 in the PAN-induced podocyte loss by affecting detachment and apoptosis in vitro-Reference-Cited by-同舟云学术

A vital role for Angptl3 in the PAN-induced podocyte loss by affecting detachment and apoptosis in vitro

Published:2015-03-29 Issue:1 Volume:16 Page:
ISSN:1471-2369
Container-title:BMC Nephrology
language:en
Short-container-title:BMC Nephrol

Author:

Dai Rufeng,Lin Yi,Liu Haimei,Rao Jia,Zhai Yihui,Zha Xiliang,Fang Xiaoyan,Xu Hong

Publisher

Springer Science and Business Media LLC

Subject

Nephrology

Link

http://link.springer.com/article/10.1186/s12882-015-0034-4/fulltext.html

Reference34 articles.

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2. Wharram BL, Goyal M, Wiggins JE, Sanden SK, Hussain S, Filipiak WE, et al. Podocyte depletion causes glomerulosclerosis: Diphtheria toxin–induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene. J Am Soc Nephrol. 2005;16:2941–52.

3. Tharaux PL, Huber TB. How many ways can a podocyte die? Semin Nephrol. 2012;32(4):394–404.

4. Shankland SJ. The podocyte’s response to injury: role in proteinuria and glomerulosclerosis. Kidney Int. 2006;69:2131–47.

5. Kriz W, Lemley KV. A potential role for mechanical forces in the detachment of podocytes and the progression of CKD. J Am Soc Nephrol. 2014;24:ASN.2014030278.

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