Acute kidney injury with extreme hyperuricemia after antithymocyte globulin treatment in a kidney transplant recipient with underlying aplastic anemia: a case report
Author:
Funder
Korean Health Technology R&D Project, Ministry for Health & Welfare, Republic of Korea
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
http://link.springer.com/content/pdf/10.1186/s12882-020-01903-9.pdf
Reference17 articles.
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2. Moreau D. Pharmacological treatment of acute renal failure in intensive care unit patients. Contrib Nephrol. 2005;147:161–73.
3. Kim YG, Huang XR, Suga S, Mazzali M, Tang D, Metz C, et al. Involvement of macrophage migration inhibitory factor (MIF) in experimental uric acid nephropathy. Mol Med. 2000;6(10):837–48.
4. Shimada M, Johnson RJ, May WS Jr, Lingegowda V, Sood P, Nakagawa T, et al. A novel role for uric acid in acute kidney injury associated with tumour lysis syndrome. Nephrol Dial Transplant. 2009;24(10):2960–4.
5. Howard SC, Jones DP, Pui CH. The tumor lysis syndrome. N Engl J Med. 2011;364(19):1844–54.
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1. Systematic review and model-based analysis to identify whether renal safety risks of URAT1 inhibitors are fully determined by uric acid-lowering efficacies;Seminars in Arthritis and Rheumatism;2023-12
2. A rare case report of reversible acute kidney injury due to hyperuricemia alone;International Urology and Nephrology;2021-01-03
3. Antithymocyte-globulin;Reactions Weekly;2020-08
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