The CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

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The CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis

Published:2012-07-12 Issue:1 Volume:9 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Ge Shujun,Shrestha Bandana,Paul Debayon,Keating Carolyn,Cone Robert,Guglielmotti Angelo,Pachter Joel S

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/1742-2094-9-171.pdf

Reference84 articles.

1. Mahad DJ, Ransohoff RM: The role of (MCP-1) CCL2 and CCR2 in multiple sclerosis and experimental autoimmune encephalomyelitis (EAE). Sem Immunol 2003, 15:23–32.

2. Eugenin EA, Osieki K, Lopez L, Goldstein H, Calderon TM, Bruce-Keller AJ, Huaser KF: CCL2/monocyte chemoattractant protein-1 mediates and enhances transmigration of human immunodeficiency virus (HIV)-infected leukocytes across the blood–brain barrier: a potential mechanism of HIV-CNS invasion and NeuroAIDS. J Neurosci 2006, 26:1098–1106.

3. Fujioka T, Purev E, Rostami A: Chemokine mRNA expression in the cauda equina of Lewis rats with experimental allergic neuritis. J Neuroimmunol 1999, 97:51–59.

4. Hickman SE, El Khoury J: Mechanisms of mononuclear phagocyte recruitment in Alzheimer’s disease. CNS Neurol Disord Drug Targets 2010, 9:168–173.

5. Chen Y, Hallenbeck JM, Ruetzler C, Bol D, Thomas K, Berman NE, Vogel SN: Overexpression of monocyte chemoattractant protein-1 in the brain exacerbates ischemic brain injury and is associated with recruitment of inflammatory cells. J Cereb Blood Flow Metab 2003, 23:748–755.

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