Sesamin ameliorates oxidative stress and mortality in kainic acid-induced status epilepticus by inhibition of MAPK and COX-2 activation-Reference-Cited by-同舟云学术

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Sesamin ameliorates oxidative stress and mortality in kainic acid-induced status epilepticus by inhibition of MAPK and COX-2 activation

Published:2011 Issue:1 Volume:8 Page:57
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:Journal of Neuroinflammation

Author:

Hsieh Peiyuan F,Hou Chien-Wei,Yao Pei-Wun,Wu Szu-Pei,Peng Yu-Fen,Shen Mei-Lin,Lin Ching-Huei,Chao Ya-Yun,Chang Ming-Hong,Jeng Kee-Ching

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Reference44 articles.

1. Pathophysiological Mechanisms of Brain Damage from Status Epilepticus

2. Emergency Treatment of Status Epilepticus: Current Thinking

3. Risk factors for complex partial seizures: A population-based case-control study

4. The CREB/CRE transcriptional pathway: protection against oxidative stress-mediated neuronal cell death

5. Rapid kindling of the hippocampus protects against neural damage resulting from status epilepticus

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