Bovine leukemia virus long terminal repeat variability: identification of single nucleotide polymorphisms in regulatory sequences
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Virology
Link
http://link.springer.com/content/pdf/10.1186/s12985-018-1062-z.pdf
Reference45 articles.
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2. Derse D. Bovine leukemia virus transcription is controlled by a virus-encoded trans-acting factor and by cis-acting response elements. J Virol. 1987;61(8):2462–71.
3. Katoh I, Kyushiki H, Sakamoto Y, Ikawa Y, Yoshinaka Y. Bovine leukemia virus matrix-associated protein MA(p15): further processing and formation of a specific complex with the dimer of the 5′-terminal genomic RNA fragment. J Virol. 1991;65(12):6845–55.
4. Sagata N, Yasunaga T, Ogawa Y, Tsuzuku-Kawamura J, Ikawa Y. Bovine leukemia virus: unique structural features of its long terminal repeats and its evolutionary relationship to human T-cell leukemia virus. Proc Natl Acad Sci U S A. 1984;81(15):4741–5.
5. Rosen CA, Sodroski JG, Kettman R, Haseltine WA. Activation of enhancer sequences in type II human T-cell leukemia virus and bovine leukemia virus long terminal repeats by virus-associated trans-acting regulatory factors. J Virol. 1986;57(3):738–44.
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