LSD1 dual function in mediating epigenetic corruption of the vitamin D signaling in prostate cancer-Reference-Cited by-同舟云学术

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LSD1 dual function in mediating epigenetic corruption of the vitamin D signaling in prostate cancer

Published:2017-08-11 Issue:1 Volume:9 Page:
ISSN:1868-7075
Container-title:Clinical Epigenetics
language:en
Short-container-title:Clin Epigenet

Author:

Battaglia Sebastiano,Karasik Ellen,Gillard Bryan,Williams Jennifer,Winchester Trisha,Moser Michael T.,Smiraglia Dominic J,Foster Barbara A.

Funder

American-Italian Cancer Foundation

U.S. Department of Defense

National Cancer Institute

National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Developmental Biology,Genetics,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13148-017-0382-y.pdf

Reference74 articles.

1. Siegel R, Ma J, Zou Z, Jemal A. Cancer statistics, 2014. CA Cancer J Clin. 2014;64(1):9–29.

2. Jones G, Strugnell SA, DeLuca HF. Current understanding of the molecular actions of vitamin D. Physiol Rev. 1998;78(4):1193–231.

3. Darwish HM, DeLuca HF. Identification of a 1,25-dihydroxyvitamin D3-response element in the 5′-flanking region of the rat calbindin D-9k gene. Proc Natl Acad Sci U S A. 1992;89(2):603–7.

4. Lam NN, Triliana R, Sawyer RK, Atkins GJ, Morris HA, O'Loughlin PD, et al. Vitamin D receptor overexpression in osteoblasts and osteocytes prevents bone loss during vitamin D-deficiency. J Steroid Biochem Mol Biol. 2014;144(Part A):128–31.

5. van de Peppel J, van Leeuwen JP. Vitamin D and gene networks in human osteoblasts. Front Physiol. 2014;5:137.

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