Increased expression of ACTH (MC2R) and androgen (AR) receptors in giant bilateral myelolipomas from patients with congenital adrenal hyperplasia-Reference-Cited by-同舟云学术

Increased expression of ACTH (MC2R) and androgen (AR) receptors in giant bilateral myelolipomas from patients with congenital adrenal hyperplasia

Published:2014-05-12 Issue:1 Volume:14 Page:
ISSN:1472-6823
Container-title:BMC Endocrine Disorders
language:en
Short-container-title:BMC Endocr Disord

Author:

Almeida Madson Q,Kaupert Laura C,Brito Luciana P,Lerario Antonio M,Mariani Beatriz M P,Ribeiro Marta,Monte Osmar,Denes Francisco T,Mendonca Berenice B,Bachega Tânia ASS

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Endocrinology, Diabetes and Metabolism

Link

http://link.springer.com/content/pdf/10.1186/1472-6823-14-42.pdf

Reference18 articles.

1. Hagiwara H, Usui T, Kimura T, Tagami T, Naruse M, Minamiguchi S, Kato T, Okuno H, Shimatsu A: Lack of ACTH and androgen receptor expression in a giant adrenal myelolipoma associated with 21-hydroxylase deficiency. Endocr Pathol. 2008, 19 (2): 122-127. 10.1007/s12022-008-9034-2.

2. Mansmann G, Lau J, Balk E, Rothberg M, Miyachi Y, Bornstein SR: The clinically inapparent adrenal mass: update in diagnosis and management. Endocr Rev. 2004, 25 (2): 309-340. 10.1210/er.2002-0031.

3. Hisamatsu H, Sakai H, Tsuda S, Shigematsu K, Kanetake H: Combined adrenal adenoma and myelolipoma in a patient with Cushing's syndrome: case report and review of the literature. Int J Urol. 2004, 11 (6): 416-418. 10.1111/j.1442-2042.2004.00815.x.

4. Maschler I, Rosenmann E, Ehrenfeld EN: Ectopic functioning adrenocortico-myelolipoma in longstanding Nelson's syndrome. Clin Endocrinol (Oxf). 1979, 10 (5): 493-497. 10.1111/j.1365-2265.1979.tb02106.x.

5. Nermoen I, Rorvik J, Holmedal SH, Hykkerud DL, Fougner KJ, Svartberg J, Husebye ES, Lovas K: High frequency of adrenal myelolipomas and testicular adrenal rest tumours in adult Norwegian patients with classical congenital adrenal hyperplasia because of 21-hydroxylase deficiency. Clin Endocrinol (Oxf). 2011, 75 (6): 753-759. 10.1111/j.1365-2265.2011.04151.x.

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