Heparin binding VEGF isoforms attenuate hyperoxic embryonic lung growth retardation via a FLK1-neuropilin-1-PKC dependent pathway-Reference-Cited by-同舟云学术

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Heparin binding VEGF isoforms attenuate hyperoxic embryonic lung growth retardation via a FLK1-neuropilin-1-PKC dependent pathway

Published:2014 Issue:1 Volume:15 Page:32
ISSN:1465-9921
Container-title:Respiratory Research
language:en
Short-container-title:Respiratory Research

Author:

Esquibies Americo E,Karihaloo Anil,Quaggin Susan E,Bazzy-Asaad Alia,Cantley Lloyd G

Publisher

Springer Science and Business Media LLC

Subject

Pulmonary and Respiratory Medicine

Reference33 articles.

1. VEGF-A signaling through Flk-1 is a critical facilitator of early embryonic lung epithelial to endothelial crosstalk and branching morphogenesis

2. Vascular Endothelial Growth Factor Induces Branching Morphogenesis/Tubulogenesis in Renal Epithelial Cells in a Neuropilin-Dependent Fashion

3. The biology of VEGF and its receptors

4. Differential expression of VEGF isoforms in mouse during development and in the adult

5. VEGF Attenuates Hyperoxic Injury through Decreased Apoptosis in Explanted Rat Embryonic Lung

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1. The Neuropilin-1/PKC axis promotes neuroendocrine differentiation and drug resistance of prostate cancer;British Journal of Cancer;2022-12-22

2. Hyperoxia-induced bronchopulmonary dysplasia: better models for better therapies;Disease Models & Mechanisms;2021-02-01

3. VEGF-B Is an Autocrine Gliotrophic Factor for Müller Cells under Pathologic Conditions;Investigative Opthalmology & Visual Science;2020-09-18

4. Stability and Species Specificity of Renal VEGF-A Splicing Patterns in Kidney Disease;PLOS ONE;2016-09-06

5. Dual Receptor Recognizing Cell Penetrating Peptide for Selective Targeting, Efficient Intratumoral Diffusion and Synthesized Anti-Glioma Therapy;Theranostics;2016

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