Upregulation of T-Type Ca2+Channels in Long-Term Diabetes Determines Increased Excitability of a Specific Type of Capsaicin-Insensitive DRG Neurons-Reference-Cited by-同舟云学术

Upregulation of T-Type Ca2+Channels in Long-Term Diabetes Determines Increased Excitability of a Specific Type of Capsaicin-Insensitive DRG Neurons

Published:2015-05-20 Issue: Volume:11 Page:s12990-015-0028
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Duzhyy Dmytro E¹,Viatchenko-Karpinski Viacheslav Y¹,Khomula Eugen V²,Voitenko Nana V¹,Belan Pavel V¹

Affiliation:

1. Department of General Physiology of the CNS and State Key Laboratory of Molecular and Cellular Biology, Bogomoletz Institute of Physiology of National Academy of Science of Ukraine, 4 Bogomoletz street, 01024 Kyiv, Ukraine

2. International Center of Molecular Physiology of National Academy of Science of Ukraine, 4 Bogomoletz street, 01024 Kyiv, Ukraine

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1186/s12990-015-0028-z

Reference61 articles.

1. Painful Diabetic Neuropathy: Epidemiology, Natural History, Early Diagnosis, and Treatment Options

2. The Diabetic Neuropathies

3. Mechanisms of Neuropathic Pain

4. In vivo silencing of the CaV3.2 T-type calcium channels in sensory neurons alleviates hyperalgesia in rats with streptozocin-induced diabetic neuropathy

5. Cell-Specific Alterations of T-Type Calcium Current in Painful Diabetic Neuropathy Enhance Excitability of Sensory Neurons

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