Small Organic Molecule Disruptors of Cav3.2 - USP5 Interactions Reverse Inflammatory and Neuropathic Pain-Reference-Cited by-同舟云学术

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Small Organic Molecule Disruptors of Cav3.2 - USP5 Interactions Reverse Inflammatory and Neuropathic Pain

Published:2015-03-14 Issue: Volume:11 Page:s12990-015-0011
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Gadotti Vinicius M¹,Caballero Agustin Garcia¹,Berger N Daniel¹,Gladding Clare M²,Chen Lina¹,Pfeifer Tom A²,Zamponi Gerald W¹

Affiliation:

1. Department of Physiology and Pharmacology, Cumming School of Medicine, University of Calgary, 3330 Hospital Drive, NW, Calgary T2N 4N1, Canada

2. Centre for Drug Research and Development, 2405 Wesbrook Mall − 4th Floor, Vancouver, BC V6T 1Z3, Canada

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1186/s12990-015-0011-8

Reference46 articles.

1. Presynaptic CaV3.2 Channels Regulate Excitatory Neurotransmission in Nociceptive Dorsal Horn Neurons

2. Regulating excitability of peripheral afferents: emerging ion channel targets

3. Regulation of T-Type Calcium Channels in the Peripheral Pain Pathway

4. Mechanical and thermal antinociception in rats following systemic administration of mibefradil, a T-type calcium channel blocker

5. Reversal of experimental neuropathic pain by T-type calcium channel blockers

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2. The Phytochemical, Quercetin, Attenuates Nociceptive and Pathological Pain: Neurophysiological Mechanisms and Therapeutic Potential;Molecules;2024-08-21

3. USP5 facilitates diabetic retinopathy development by stabilizing ROBO4 via deubiquitination;Cellular Signalling;2024-08

4. Unraveling the Immune Regulatory Functions of USP5: Implications for Disease Therapy;Biomolecules;2024-06-12

5. α-Synuclein oligomers potentiate neuroinflammatory NF-κB activity and induce Cav3.2 calcium signaling in astrocytes;Translational Neurodegeneration;2024-02-21

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