Pathogenesis of bronchopulmonary dysplasia: when inflammation meets organ development
Author:
Funder
UKGM Forschungsförderung
Von-Behring-Röntgen-Stiftung
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
http://link.springer.com/content/pdf/10.1186/s40348-016-0051-9.pdf
Reference72 articles.
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2. Islam JA, Keller RL, Aschner JL, Hartert TV, Moore PE et al (2015) Understanding the Short- and Long-Term Respiratory Outcomes of Prematurity and Bronchopulmonary Dysplasia. Am J Respir Crit Care Med 192(2):134–156. doi: 10.1164/rccm.201412-2142PP
3. Didon L, Roos AB, Elmberger GP, Gonzalez FJ, Nord M et al (2010) Lung-specific inactivation of CCAAT/enhancer binding protein a causes a pathological pattern characteristic of COPD. Eur Respir J 35(1):186–197. doi: 10.1183/09031936.00185008
4. Carraro S, Giordano G, Pirillo P, Maretti M, Reniero F, Cogo PE, Perilongo G, Stocchero M, Baraldi E et al (2015) Airway metabolic anomalies in adolescents with bronchopulmonary dysplasia: new insights from the metabolomic approach. J Pediatr 166(2):234–239. doi: 10.1016/j.jpeds.2014.08.049
5. Suursalmi P, Kopeli T, Korhonen P, Lehtimäki L, Nieminen R, Luukkaala T, Moilanen E, Korppi M, Paassilta M, Tammela O et al (2015) Very low birthweight bronchopulmonary dysplasia survivors show no substantial association between lung function and current inflammatory markers. Acta Paediatr 104(3):264–268. doi: 10.1111/apa.12837
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