Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome-Reference-Cited by-同舟云学术

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Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome

Published:2012 Issue:1 Volume:14 Page:R40
ISSN:1478-6354
Container-title:Arthritis Research & Therapy
language:en
Short-container-title:Arthritis Res Ther

Author:

Yin Hongen,Nguyen Cuong Q,Samuni Yuval,Uede Toshimitsu,Peck Ammon B,Chiorini John A

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy,Rheumatology

Reference26 articles.

1. Pathogenetic mechanisms in the initiation and perpetuation of Sjögren's syndrome

2. CD4 cytotoxic and dendritic cells in the immunopathologic lesion of Sjögren’s syndrome

3. Abatacept for Rheumatoid Arthritis Refractory to Tumor Necrosis Factor α Inhibition

4. Interleukin-12 induces salivary gland dysfunction in transgenic mice, providing a new model of SjÃ¶gren's syndrome

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1. Salivary gland biopsy as a prognostic tool in Sjögren’s syndrome;Expert Review of Clinical Immunology;2024-06-24

2. Correction of LAMP3-associated salivary gland hypofunction by aquaporin gene therapy;Scientific Reports;2022-11-03

3. An overview of immune checkpoint therapy in autoimmune diseases;International Immunopharmacology;2022-06

4. Stimulation of regulatory T cells with Lactococcus lactis expressing enterotoxigenic E. coli colonization factor antigen 1 retains salivary flow in a genetic model of Sjögren’s syndrome;Arthritis Research & Therapy;2021-04-06

5. Gene Therapy for Sjögren’s Syndrome;Sjögren's Syndrome and Oral Health;2021

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