Author:
Mosad Eman,Hamed Hosny B,Bakry Rania M,Ezz-Eldin Azza M,Khalifa Nesrine M
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology,Molecular Biology,Hematology
Reference30 articles.
1. Endo C, Oda M, Nishiuchi R, Seino Y: Persistence of TEL-AML1 transcript in acute lymphoblastic leukemia in long-term remission. Pediatr Int. 2003, 45: 275-280. 10.1046/j.1442-200X.2003.01709.x.
2. McLean TW, Ringold S, Neuberg D, Stegmaier K, Tantravahi R, Ritz J, Koeffler HP, Takeuchi S, Janssen JW, Seriu T, Bartram CR, Sallan SE, Gilliland DG, Golub TR: TEL-AML1 dimerizes and is associated with a favorable outcome in childhood acute lymphoblastic leukemia. Blood. 1996, 88: 4252-4258.
3. Eguchi-Ishimae M, Eguchi M, Tanaka K, Hamamoto K, Ohki M, Ueda K, Kamada N: Fluorescence in situ hybridization analysis of 12;21 translocation in Japanese childhood acute lymphoblastic leukemia. Jpn J Cancer Res. 1998, 89: 783-788.
4. Loh ML, Silverman LB, Young ML, Neuberg D, Golub TR, Sallan SE, Gilliland DG: Incidence of TEL/AML1 fusion in children with relapsed acute lymphoblastic leukemia. Blood. 1998, 92: 4792-4797.
5. Shurtleff SA, Buijs A, Behm FG, Rubnitz JE, Raimondi SC, Hancock ML, Chan GC, Pui CH, Grosveld G, Downing JR: TEL-AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis. Leukemia. 1995, 9 (12): 985-989.
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