P53 codon 72 polymorphism, human papillomavirus infection, and their interaction to oral carcinoma susceptibility-Reference-Cited by-同舟云学术

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P53 codon 72 polymorphism, human papillomavirus infection, and their interaction to oral carcinoma susceptibility

Published:2015-06-30 Issue:1 Volume:16 Page:
ISSN:1471-2156
Container-title:BMC Genetics
language:en
Short-container-title:BMC Genet

Author:

Hou Jun,Gu Ying,Hou Wei,Wu Song,Lou Yin,Yang Wenyu,Zhu Ling,Hu Yukun,Sun Ming,Xue Haowei

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics

Link

http://link.springer.com/article/10.1186/s12863-015-0235-7/fulltext.html

Reference38 articles.

1. Tanaka T, Tanaka M, Tanaka T. Oral carcinogenesis and oral cancer chemoprevention: a review. Patholog Res Int. 2011;2011:431246.

2. Petersen PE. Oral cancer prevention and control—the approach of the World Health Organization. Oral Oncol. 2009;45:454–60.

3. Zain RB, Ikeda N, Gupta PC, Warnakulasuriya S, van Wyk CW, Shrestha P, et al. Oral mucosal lesions associated with betel quid, areca nut and tobacco chewing habits: consensus from a workshop held in Kuala Lumpur, Malaysia, November 25–27, 1996. J Oral Pathol Med. 1999;28:1–4.

4. Pfeifer GP, Denissenko MF, Olivier M, Tretyakova N, Hecht SS, Hainaut P. Tobacco smoke carcinogens, DNA damage and p53 mutations in smoking-associated cancers. Oncogene. 2002;21:7435–51.

5. Zhong Y, Carmella SG, Upadhyaya P, Hochalter JB, Rauch D, Oliver A, et al. Immediate consequences of cigarette smoking: rapid formation of polycyclic aromatic hydrocarbon diol epoxides. Chem Res Toxicol. 2011;24:246–52.

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