Does rituximab improve clinical outcomes of patients with thyroid-associated ophthalmopathy? A systematic review and meta-analysis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Ophthalmology,General Medicine
Link
http://link.springer.com/content/pdf/10.1186/s12886-018-0679-4.pdf
Reference31 articles.
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2. Smith TJ. Pathogenesis of graves’ orbitopathy: a 2010 update. J Endocrinol Investig. 2010;33(6):414–21.
3. Smith TJ, Tsai CC, Shih MJ, Tsui S, Chen B, Han R, et al. Unique attributes of orbital fibroblasts and global alterations in IGF-1 receptor signaling could explain thyroid-associated Ophthalmopathy. Thyroid Official J Am Thyroid Assoc. 2008;18(9):983.
4. Douglas RS, Naik V, Hwang CJ, Afifiyan NF, Gianoukakis AG, Sand D, et al. B cells from patients with Graves' disease aberrantly express the IGF-1 receptor: implications for disease pathogenesis. J Immunol. 2008;181(8):5768.
5. Bartalena L, Lai A, Compri E, Marcocci C, Tanda ML. Novel immunomodulating agents for graves orbitopathy. Ophthalmic Plast Reconstr Surg. 2008;24(4):251.
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1. Potential role of IGF-1R in the interaction between orbital fibroblasts and B lymphocytes: an implication for B lymphocyte depletion in the active inflammatory phase of thyroid-associated ophthalmopathy;BMC Immunology;2024-05-11
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3. Monoclonal antibodies for the treatment of Graves’ ophthalmopathy: A systematic review and meta-analysis;Saudi Journal of Ophthalmology;2023-04
4. Rituximab for thyroid-associated ophthalmopathy;Cochrane Database of Systematic Reviews;2022-06-16
5. 2022 Update on Clinical Management of Graves Disease and Thyroid Eye Disease;Endocrinology and Metabolism Clinics of North America;2022-06
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