Upstream molecular signaling pathways of p27(Kip1) expression in human breast cancer cells in vitro: differential effects of 4-hydroxytamoxifen and deficiency of either D-(+)-glucose or L-leucine-Reference-Cited by-同舟云学术

Upstream molecular signaling pathways of p27(Kip1) expression in human breast cancer cells in vitro: differential effects of 4-hydroxytamoxifen and deficiency of either D-(+)-glucose or L-leucine

Published:2011 Issue:1 Volume:11 Page:31
ISSN:1475-2867
Container-title:Cancer Cell International
language:en
Short-container-title:Cancer Cell International

Author:

Eto Isao

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Reference32 articles.

1. Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids

2. Cell cycle-dependent translation of p27 involves a responsive element in its 5'-UTR that overlaps with a uORF

3. Translational Control of p27 Kip1 Accumulation During the Cell Cycle

4. Enhanced Ribosomal Association of p27Kip1 mRNA Is a Mechanism Contributing to Accumulation during Growth Arrest

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5. Expression of p27Kip1, a cell cycle repressor protein, is inversely associated with potential carcinogenic risk in the genetic rodent models of obesity and long-lived Ames dwarf mice;Metabolism;2013-06