The magnitude of nephron number reduction mediates intrauterine growth-restriction-induced long term chronic renal disease in the rat. A comparative study in two experimental models-Reference-Cited by-同舟云学术

The magnitude of nephron number reduction mediates intrauterine growth-restriction-induced long term chronic renal disease in the rat. A comparative study in two experimental models

Published:2016-11-30 Issue:1 Volume:14 Page:
ISSN:1479-5876
Container-title:Journal of Translational Medicine
language:en
Short-container-title:J Transl Med

Author:

Boubred Farid^ORCID,Daniel Laurent,Buffat Christophe,Tsimaratos Michel,Oliver Charles,Lelièvre-Pégorier Martine,Simeoni Umberto

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1186/s12967-016-1086-3.pdf

Reference41 articles.

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2. Vehaskari VM, Woods LL. Prenatal programming of hypertension: lessons from experimental models. J Am Soc Nephrol. 2005;16:254–6.

3. Gluckman PD, Hanson MA, Cooper C, Thornburg KL. Effect of in utero and early-life conditions on adult health diseases. N Engl J Med. 2008;359:61–73.

4. Coresh J, Selvin E, Stevens LA, et al. Prevalence of chronic kidney disease in the United States. J Am Med Assoc. 2007;298:2038–47.

5. McDonald TJ, Franko KL, Brown JM, Jenkins SL, Nathanielsz PW, Nijland MJ. Betamethasone in the last week of pregnancy causes fetal growth retardation but nor adult hypertension in rats. J Soc Gynecol Invest. 2003;10:469–73.

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