Enhancing energy and glucose metabolism by disrupting triglyceride synthesis: Lessons from mice lacking DGAT1
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nutrition and Dietetics,Endocrinology, Diabetes and Metabolism,Medicine (miscellaneous)
Link
http://link.springer.com/content/pdf/10.1186/1743-7075-3-10.pdf
Reference16 articles.
1. Smith SJ, Cases S, Jensen DR, Chen HC, Sande E, Tow B, Sanan DA, Raber J, Eckel RH, Farese RV: Obesity resistance and multiple mechanisms of triglyceride synthesis in mice lacking DGAT. Nat Genet. 2000, 25: 87-90. 10.1038/75651.
2. Chen HC, Farese RV: DGAT and triglyceride synthesis: A new target for obesity treatment?. Trends Cardiovasc Me. 2000, 10: 188-192. 10.1016/S1050-1738(00)00066-9.
3. Yen CL, Monetti M, Burri BJ, Farese RV: The triacylglycerol synthesis enzyme DGAT1 also catalyzes the synthesis of diacylglycerols, waxes, and retinyl esters. J Lipid Res. 2005, 46: 1502-1511. 10.1194/jlr.M500036-JLR200.
4. Chen HC, Smith SJ, Ladha Z, Jensen DR, Ferreira LD, Pulawa LK, McGuire JG, Pitas RE, Eckel RH, Farese RV: Increased insulin and leptin sensitivity in mice lacking acyl CoA:diacylglycerol acyltransferase 1. J Clin Invest. 2002, 109: 1049-1055. 10.1172/JCI200214672.
5. Cao J, Burn P, Shi Y: Properties of the mouse intestinal acyl-CoA:monoacylglycerol acyltransferase, MGAT2. J Biol Chem. 2003, 278: 25657-25663. 10.1074/jbc.M302835200.
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