Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells-Reference-Cited by-同舟云学术

Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells

Published:2018-06-08 Issue:1 Volume:20 Page:
ISSN:1478-6362
Container-title:Arthritis Research & Therapy
language:en
Short-container-title:Arthritis Res Ther

Author:

Talbot Jhimmy,Peres Raphael S.,Pinto Larissa G.,Oliveira Rene D. R.,Lima Kalil A.,Donate Paula B.,Silva Jaqueline R.,Ryffel Bernard,Cunha Thiago M.,Alves-Filho José C.,Liew Foo Y.,Louzada-Junior Paulo,de Queiroz Cunha Fernando

Funder

Fundação de Amparo à Pesquisa do Estado de São Paulo

Center for Research in Inflammatory Disease

European Union Seventh Framework Programme

Publisher

Springer Science and Business Media LLC

Link

http://link.springer.com/content/pdf/10.1186/s13075-018-1609-9.pdf

Reference57 articles.

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3. Firestein GS. Evolving concepts of rheumatoid arthritis. Nature. 2003;423(6937):356–61.

4. Sugiyama D, Nishimura K, Tamaki K, Tsuji G, Nakazawa T, Morinobu A, Kumagai S. Impact of smoking as a risk factor for developing rheumatoid arthritis: a meta-analysis of observational studies. Ann Rheum Dis. 2010;69(1):70–81.

5. Saevarsdottir S, Rezaei H, Geborek P, Petersson I, Ernestam S, Albertsson K, Forslind K, van Vollenhoven RF, SWEFOT study group. Current smoking status is a strong predictor of radiographic progression in early rheumatoid arthritis: results from the SWEFOT trial. Ann Rheum Dis. 2015;74(8):1509–14.

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