Ketamine exacerbates cortical neuroapoptosis under hyperoxic conditions by upregulating expression of the N-methyl-D-aspartate receptor subunit NR1 in the developing rat brain-Reference-Cited by-同舟云学术

Ketamine exacerbates cortical neuroapoptosis under hyperoxic conditions by upregulating expression of the N-methyl-D-aspartate receptor subunit NR1 in the developing rat brain

Published:2018-05-10 Issue:1 Volume:18 Page:
ISSN:1471-2253
Container-title:BMC Anesthesiology
language:en
Short-container-title:BMC Anesthesiol

Author:

Wu Changyi,Wang Jun,Guo Xiangyang,Zhang Ying^ORCID

Funder

the National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Anesthesiology and Pain Medicine

Link

http://link.springer.com/content/pdf/10.1186/s12871-018-0511-y.pdf

Reference21 articles.

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2. Paule MG, Li M, Allen RR, Liu F, Zou X, Hotchkiss C, et al. Ketamine anesthesia during the first week of life can cause long-lasting cognitive deficits in rhesus monkeys. Neurotoxicol Teratol. 2011;33:220–30.

3. Scallet AC, Schmued LC, Slikker W Jr, Grunberg N, Faustino PJ, Davis H, et al. Developmental neurotoxicity of ketamine: morphometric confirmation, exposure parameters, and multiple fluorescent labeling of apoptotic neurons. Toxicol Sci. 2004;81:364–70.

4. Arkovitz MS, Szabo C, Garcia VF, Wong HR, Wispe JR. Differential effects of hyperoxia on the inducible and constitutive isoforms of nitric oxide synthase in the lung. Shock. 1997;7:345–50.

5. Sennlaub F, Courtois Y, Goureau O. Inducible nitric oxide synthase mediates the change from retinal to vitreal neovascularization in ischemic retinopathy. J Clin Invest. 2001;107:717–25.

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