Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury-Reference-Cited by-同舟云学术

Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury

Published:2018-04-04 Issue:1 Volume:13 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Main Bevan S.,Villapol Sonia,Sloley Stephanie S.,Barton David J.,Parsadanian Maia,Agbaegbu Chinyere,Stefos Kathryn,McCann Mondona S.,Washington Patricia M.,Rodriguez Olga C.,Burns Mark P.

Funder

National Institute of Neurological Disorders and Stroke

U.S. Department of Health and Human Services

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-018-0249-5.pdf

Reference87 articles.

1. Neuwelt E, et al. Strategies to advance translational research into brain barriers. The Lancet Neurology. 2008;7(1):84–96.

2. Shlosberg D, et al. Blood-brain barrier breakdown as a therapeutic target in traumatic brain injury. Nat Rev Neurol. 2010;6(7):393–403.

3. Mahley RW. Apolipoprotein E: cholesterol transport protein with expanding role in cell biology. Science. 1988;240(4852):622–30.

4. Chen Y, et al. Motor and cognitive deficits in apolipoprotein E-deficient mice after closed head injury. Neuroscience. 1997;80(4):1255–62.

5. Namjoshi DR, et al. The liver X receptor agonist GW3965 improves recovery from mild repetitive traumatic brain injury in mice partly through apolipoprotein E. PLoS One. 2013;8(1):e53529.

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