Insights from new in vivo models of TREM2 variants
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology
Link
https://link.springer.com/content/pdf/10.1186/s13024-023-00609-4.pdf
Reference12 articles.
1. Guerreiro R, Wojtas A, Bras J, Carrasquillo M, Rogaeva E, Majounie E, et al. TREM2 variants in Alzheimer’s disease. N Engl J Med. 2013;368(2):117–27.
2. Wang Y, Cella M, Mallinson K, Ulrich JD, Young KL, Robinette ML, et al. TREM2 lipid sensing sustains the microglial response in an Alzheimer’s disease model. Cell. 2015;160(6):1061–71.
3. Tran KM, Kawauchi S, Kramar EA, Rezaie N, Liang HY, Sakr JS, et al. A Trem 2(R47H) mouse model without cryptic splicing drives age- and disease-dependent tissue damage and synaptic loss in response to plaques. Mol Neurodegener. 2023;18(1):12.
4. Qiao W, Chen Y, Zhong J, Madden BJ, Charlesworth CM, Martens YA, et al. Trem2 H157Y increases soluble TREM2 production and reduces amyloid pathology. Mol Neurodegener. 2023;18(1):8.
5. Cheng-Hathaway PJ, Reed-Geaghan EG, Jay TR, Casali BT, Bemiller SM, Puntambekar SS, et al. The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer’s disease. Mol Neurodegener. 2018;13(1):29.
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1. Generation of sophisticated Alzheimer’s disease mouse models and research advances utilizing them;Translational and Regulatory Sciences;2023
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