AMPA-ergic regulation of amyloid-β levels in an Alzheimer’s disease mouse model-Reference-Cited by-同舟云学术

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AMPA-ergic regulation of amyloid-β levels in an Alzheimer’s disease mouse model

Published:2018-05-15 Issue:1 Volume:13 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Hettinger Jane C.,Lee Hyo,Bu Guojun,Holtzman David M.,Cirrito John R.^ORCID

Funder

National Institute of Neurological Disorders and Stroke

National Institute on Aging

Charles F. and Joanne Knight ADRC at Washington University

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-018-0256-6.pdf

Reference80 articles.

1. Sperling RA, Aisen PS, Beckett LA, Bennett DA, Craft S, Fagan AM, et al. Toward defining the preclinical stages of Alzheimer ’ s disease : recommendations from the National Institute on Aging and the Alzheimer ’ s association workgroup. Alzheimers Dement. 2011;7:280-92.

2. Hardy JA, Higgins GA. Alzheimer’s disease: the amyloid cascade hypothesis. Science. 1992;256:184–5.

3. Musiek ES, Holtzman DM. Three dimensions of the amyloid hypothesis: time, space and “wingmen”. Nat Neurosci. 2015;18:800–6. Nature Research

4. Bero AW, Yan P, Roh JH, Cirrito JR, Stewart FR, Raichle ME, et al. Neuronal activity regulates the regional vulnerability to amyloid-β deposition. Nat Neurosci. 2011;14(6):750. Nature Research

5. Meyer-Luehmann M, Stalder M, Herzig MC, Kaeser SA, Kohler E, Pfeifer M, et al. Extracellular amyloid formation and associated pathology in neural grafts. Nat Neurosci. 2003;6:370–7. Nature Publishing Group

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