Late onset Alzheimer’s disease genetics implicates microglial pathways in disease risk-Reference-Cited by-同舟云学术

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Late onset Alzheimer’s disease genetics implicates microglial pathways in disease risk

Published:2017-05-26 Issue:1 Volume:12 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Efthymiou Anastasia G.,Goate Alison M.

Funder

National Institutes of Health

JPB Foundation

Foundation for the National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-017-0184-x.pdf

Reference98 articles.

1. Alzheimer’s Association. 2016 Alzheimer’s Disease Facts and Figures. Alzheimers Dement. 2016;12(4).

2. Goate A, et al. Segregation of a missense mutation in the amyloid precursor protein. Lett to Nat. 1991;349:704–6.

3. Sherrington R, et al. Cloning of a gene bearing missense mutations in early-onset familial Alzheimer’s disease. Nature. 1995;375:754–60.

4. Alzheimer’s Disease Collaborative Group. The structure of the presenilin 1 (S182) gene and identification of six novel mutations in early onset AD families. Nat Genet. 1995;11:219–22.

5. Rogaev EI, et al. Familial Alzheimer’s disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer’s disease type 3 gene. Nature. 1995;376:775–8.

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