A proteomic analysis of LRRK2 binding partners reveals interactions with multiple signaling components of the WNT/PCP pathway-Reference-Cited by-同舟云学术

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A proteomic analysis of LRRK2 binding partners reveals interactions with multiple signaling components of the WNT/PCP pathway

Published:2017-07-11 Issue:1 Volume:12 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Salašová Alena,Yokota Chika,Potěšil David,Zdráhal Zbyněk,Bryja Vítězslav,Arenas Ernest^ORCID

Funder

Vetenskapsrådet (SE)

Swedish Foundation for Strategic Research

Seventh Framework Programme

Karolinska Institutet

CIISB

CEITEC 2020

Czech Science Foundation

Cancerfonden

Hjärnfonden

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-017-0193-9.pdf

Reference123 articles.

1. Lin MK, Farrer MJ. Genetics and genomics of Parkinson's disease. Genome Med. 2014;6:48.

2. Dachsel JC, Nishioka K, Vilarino-Guell C, Lincoln SJ, Soto-Ortolaza AI, Kachergus J, et al. Heterodimerization of Lrrk1-Lrrk2: implications for LRRK2-associated Parkinson disease. Mech Ageing Dev. 2010;131:210–4.

3. Klein C, Westenberger A. Genetics of Parkinson's disease. Cold Spring Harb Perspect Med. 2012;2:a008888.

4. Wood-Kaczmar A, Gandhi S, Wood NW. Understanding the molecular causes of Parkinson's disease. Trends Mol Med. 2006;12:521–8.

5. Kumaran R, Cookson MR. Pathways to parkinsonism Redux: convergent pathobiological mechanisms in genetics of Parkinson's disease. Hum Mol Genet. 2015;24:R32–44.

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