Synaptic dysfunction and septin protein family members in neurodegenerative diseases-Reference-Cited by-同舟云学术

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Synaptic dysfunction and septin protein family members in neurodegenerative diseases

Published:2015-04-03 Issue:1 Volume:10 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Marttinen Mikael,Kurkinen Kaisa MA,Soininen Hilkka,Haapasalo Annakaisa,Hiltunen Mikko

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-015-0013-z.pdf

Reference129 articles.

1. Cooke SF, Bliss TV. Plasticity in the human central nervous system. Brain. 2006;129:1659–73.

2. Eidelberg D, Surmeier DJ. Brain networks in Huntington disease. J Clin Invest. 2011;121:484–92.

3. Goldberg MS, Pisani A, Haburcak M, Vortherms TA, Kitada T, Costa C, et al. Nigrostriatal dopaminergic deficits and hypokinesia caused by inactivation of the familial Parkinsonism-linked gene DJ-1. Neuron. 2005;45:489–96.

4. Li S, Hong S, Shepardson NE, Walsh DM, Shankar GM, Selkoe D. Soluble oligomers of amyloid Beta protein facilitate hippocampal long-term depression by disrupting neuronal glutamate uptake. Neuron. 2009;62:788–801.

5. Oh MC, Derkach VA, Guire ES, Soderling TR. Extrasynaptic membrane trafficking regulated by GluR1 serine 845 phosphorylation primes AMPA receptors for long-term potentiation. J Biol Chem. 2006;281:752–8.

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