Sepsis protects the myocardium and other organs from subsequent ischaemic/reperfusion injury via a MAPK-dependent mechanism
Author:
Publisher
Springer Science and Business Media LLC
Subject
Critical Care and Intensive Care Medicine
Link
http://link.springer.com/content/pdf/10.1186/s40635-014-0035-9.pdf
Reference47 articles.
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2. Mueller-Werdan U, Reithmann C, Werdan K: Cytokines and the heart: molecular mechanisms of septic cardiomyopathy. Springer, Berlin; 1996.
3. Müller-Werdan U, Werdan K: Septic cardiomyopathy. Curr Opin Crit Care 1999, 5: 415–421. 10.1097/00075198-199910000-00013
4. Chao W, Shen Y, Zhu X, Zhao H, Novikov M, Schmidt U, Rosenzweig A: Lipopolysaccharide improves cardiomyocyte survival and function after serum deprivation. J Biol Chem 2005,280(23):21997–22005. 10.1074/jbc.M413676200
5. Fauvel H, Marchetti P, Chopin C, Formstecher P, Nevière R: Differential effects of caspase inhibitors on endotoxin-induced myocardial dysfunction and heart apoptosis. Am J Physiol Heart Circ Physiol 2001,280(4):H1608-H1614.
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