The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA

Author:

Maurer-Stroh Sebastian,Lee Raphael TC,Gunalan Vithiagaran,Eisenhaber Frank

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Virology

Reference40 articles.

1. Lebarbenchon C, Stallknecht DE: Host shifts and molecular evolution of H7 avian influenza virus hemagglutinin. Virol J 2011, 8: 328. 10.1186/1743-422X-8-328

2. Gambaryan AS, Matrosovich TY, Philipp J, Munster VJ, Fouchier RAM, Cattoli G, Capua I, Krauss SL, Webster RG, Banks J, Bovin NV, Klenk H-D, Matrosovich MN: Receptor-binding profiles of H7 subtype influenza viruses in different host species. J Virol 2012, 86: 4370-4379. 10.1128/JVI.06959-11

3. Highly pathogenic avian influenza, Mexico. Follow-up report No. 1. Information received on 26/06/2012 from Dr Hugo fragoso sánchez, director general de salud animal, SENASICA, SAGARPA, Mexico. http://www.oie.int/wahis_2/public/wahid.php/Reviewreport/Review?reportid=12074

4. FAO: Highly pathogenic avian influenza in Mexico (H7N3) - a significant threat to poultry production not to be underestimated. EMPRES WATCH 2012., 26: http://www.fao.org/docrep/016/an395e/an395e.pdf

5. Notes from the field: highly pathogenic avian influenza a (H7N3) virus infection in Two poultry workers — Jalisco, Mexico, July 2012. [ http://www.cdc.gov/mmwr/preview/mmwrhtml/mm6136a4.htm?s_cid=mm6136a4_e ]

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