NaCl pretreatment attenuates H.pylori-induced DNA damage and exacerbates proliferation of gastric epithelial cells (GES-1)
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Infectious Diseases,Oncology,Epidemiology
Link
http://link.springer.com/content/pdf/10.1186/s13027-015-0003-3.pdf
Reference37 articles.
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2. Azuma T. Helicobacter pylori CagA protein variation associated with gastric cancer in Asia. J Gastroenterol. 2004;39:97–103.
3. Ishii Y, Umemura T, Kanki K. Possible involvement of NO-mediated oxidative stress in induction of rat forestomach damage and cell proliferation by combined treatment with catechol and sodium nitrite. Arch Biochem Biophys. 2006;447:127–35.
4. Kuroiwa Y, Ishii Y, Umemura T. Combined treatment with green tea catechins and sodium nitrite selectively promotes rat forestomach carcinogenesis after initiation with N-methyl-N’-nitro-N-nitrosoguanidine. Cancer Sci. 2007;98:949–57.
5. Uehara T, Ma D, Yao Y. H.pylori infection is associated with DNA damage of Lgr5-positive epithelial stem cells in the stomach of patients with gastric cancer. Dig Dis Sci. 2013;58:140–9.
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