Association of HLA-G 3′ UTR polymorphism and expression with the progression of cervical lesions in human papillomavirus 18 infections-Reference-Cited by-同舟云学术

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Association of HLA-G 3′ UTR polymorphism and expression with the progression of cervical lesions in human papillomavirus 18 infections

Published:2018-12 Issue:1 Volume:13 Page:
ISSN:1750-9378
Container-title:Infectious Agents and Cancer
language:en
Short-container-title:Infect Agents Cancer

Author:

Xu Hui-Hui,Zhang Xia,Zheng Hai-Hong,Han Qiu-Yue,Lin Ai-Fen,Yan Wei-Hua^ORCID

Funder

National Natural Science Foundation of China

Zhejiang Provincial Key Laboratory of Wood Science and Technology

the Health Bureau of Zhejiang Province

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Infectious Diseases,Oncology,Epidemiology

Link

http://link.springer.com/content/pdf/10.1186/s13027-018-0217-2.pdf

Reference39 articles.

1. Torre LA, Bray F, Siegel RL, Ferlay J, Lortet-Tieulent J, Jemal A. Global cancer statistics, 2012. CA Cancer J Clin. 2015;65:87–108.

2. Crosbie EJ, Einstein MH, Franceschi S, Kitchener HC. Human papillomavirus and cervical cancer. Lancet. 2013;382:889–99.

3. Sasagawa T, Takagi H, Makinoda S. Immune responses against human papillomavirus (HPV) infection and evasion of host defense in cervical cancer. J Infect Chemother. 2012;18:807–15.

4. Carosella ED, HoWangYin KY, Favier B, LeMaoult J. HLA-G-dependent suppressor cells: diverse by nature, function, and significance. Hum Immunol. 2008;69:700–7.

5. Yan WH. HLA-G Expression in Cancers: Potential Role in Diagnosis, Prognosis and Therapy. Endocr Metab Immune Disord Drug Targets. 2011;11:76–89.

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