C-Cbl negatively regulates TRAF6-mediated NF-κB activation by promoting K48-linked polyubiquitination of TRAF6-Reference-Cited by-同舟云学术

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C-Cbl negatively regulates TRAF6-mediated NF-κB activation by promoting K48-linked polyubiquitination of TRAF6

Published:2019-05-14 Issue:1 Volume:24 Page:
ISSN:1425-8153
Container-title:Cellular & Molecular Biology Letters
language:en
Short-container-title:Cell Mol Biol Lett

Author:

Jang Hyun-Duk,Hwang Hye Zin,Kim Hyo-Soo,Lee Soo Young

Funder

Korea Health Industry Development Institute

National Research Foundation of Korea

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://link.springer.com/content/pdf/10.1186/s11658-019-0156-y.pdf

Reference31 articles.

1. Manthiram K, Zhou Q, Aksentijevich I, Kastner DL. The monogenic autoinflammatory diseases define new pathways in human innate immunity and inflammation. Nat Immunol. 2017;18(8):832–42.

2. Deng L, Wang C, Spencer E, Yang L, Braun A, You J, et al. Activation of the IkappaB kinase complex by TRAF6 requires a dimeric ubiquitin-conjugating enzyme complex and a unique polyubiquitin chain. Cell. 2000;103(2):351–61.

3. Locksley RM, Killeen N, Lenardo MJ. The TNF and TNF receptor superfamilies: integrating mammalian biology. Cell. 2001;104(4):487–501.

4. Wang C, Deng L, Hong M, Akkaraju GR, Inoue J, Chen ZJ. TAK1 is a ubiquitin-dependent kinase of MKK and IKK. Nature. 2001;412(6844):346–51.

5. Rudd CE, Schneider H. Lymphocyte signaling: Cbl sets the threshold for autoimmunity. Curr Biol. 2000;10(9):R344–7.

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