Tumor-associated macrophage-derived itaconic acid contributes to nasopharyngeal carcinoma progression by promoting immune escape via TET2-Reference-Cited by-同舟云学术

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Tumor-associated macrophage-derived itaconic acid contributes to nasopharyngeal carcinoma progression by promoting immune escape via TET2

Published:2024-08-27 Issue:1 Volume:22 Page:
ISSN:1478-811X
Container-title:Cell Communication and Signaling
language:en
Short-container-title:Cell Commun Signal

Author:

Zhang Xiaowei,Qian Shen’er,Wu Ping’an,Yu Benquan,Yin Danhui,Peng Xia,Li Shisheng,Xiao Zian,Xie Zuozhong

Funder

Natural Science Foundation of Hunan Province

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1186/s12964-024-01799-0.pdf

Reference38 articles.

1. Bossi P, Chan AT, Licitra L, Trama A, Orlandi E, Hui EP, et al. Nasopharyngeal carcinoma: ESMO-EURACAN clinical practice guidelines for diagnosis, treatment and follow-up(†). Ann Oncol. 2021;32(4):452–65.

2. Chiang CL, Guo Q, Ng WT, Lin S, Ma TSW, Xu Z, et al. Prognostic factors for overall survival in nasopharyngeal Cancer and implication for TNM staging by UICC: a systematic review of the literature. Front Oncol. 2021;11:703995.

3. de Visser KE, Joyce JA. The evolving tumor microenvironment: from cancer initiation to metastatic outgrowth. Cancer Cell. 2023;41(3):374–403.

4. Joyce JA, Fearon DT. T cell exclusion, immune privilege, and the tumor microenvironment. Science. 2015;348(6230):74–80.

5. Xiang X, Wang J, Lu D, Xu X. Targeting tumor-associated macrophages to synergize tumor immunotherapy. Signal Transduct Target Ther. 2021;6(1):75.

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