Oxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout mice-Reference-Cited by-同舟云学术

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Oxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout mice

Published:2015-04-18 Issue:1 Volume:5 Page:
ISSN:2044-5040
Container-title:Skeletal Muscle
language:en
Short-container-title:Skeletal Muscle

Author:

Paolini Cecilia,Quarta Marco,Wei-LaPierre Lan,Michelucci Antonio,Nori Alessandra,Reggiani Carlo,Dirksen Robert T,Protasi Feliciano

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Orthopedics and Sports Medicine

Link

http://link.springer.com/content/pdf/10.1186/s13395-015-0035-9.pdf

Reference62 articles.

1. Schneider MF. Control of calcium release in functioning skeletal muscle fibers. Annu Rev Physiol. 1994;56:463–84.

2. Dulhunty AF, Beard NA, Pouliquin P, Kimura T. Novel regulators of RyR Ca2+ release channels: insight into molecular changes in genetically-linked myopathies. J Muscle Res Cell Motil. 2006;27(5–7):351–65.

3. Franzini-Armstrong C, Protasi F. Ryanodine receptors of striated muscles: a complex channel capable of multiple interactions. Physiol Rev. 1997;77(3):699–729.

4. Zhang L, Kelley J, Schmeisser G, Kobayashi YM, Jones LR. Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor: proteins of the cardiac junctional sarcoplasmic reticulum membrane. J Biol Chem. 1997;272(37):23389–97.

5. Maclennan DH, Zvaritch E. Mechanistic models for muscle diseases and disorders originating in the sarcoplasmic reticulum. Biochim Biophys Acta. 2011;1813(5):948–64. doi:10.1016/j.bbamcr.2010.11.009.

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