The lysine methyltransferase Ehmt2/G9a is dispensable for skeletal muscle development and regeneration

Author:

Zhang Regan-Heng,Judson Robert N.,Liu David Y.,Kast Jürgen,Rossi Fabio M. V.

Funder

Canadian Institutes of Health Research

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Orthopedics and Sports Medicine

Reference50 articles.

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3. Gustafsson MK, Pan H, Pinney DF, Liu Y, Lewandowski A, Epstein DJ, et al. Myf5 is a direct target of long-range Shh signaling and Gli regulation for muscle specification. Genes Dev. 2002;16(1):114–26.

4. Tajbakhsh S, Borello U, Vivarelli E, Kelly R, Papkoff J, Duprez D, et al. Differential activation of Myf5 and MyoD by different Wnts in explants of mouse paraxial mesoderm and the later activation of myogenesis in the absence of Myf5. Development. 1998;125(21):4155–62.

5. Sartorelli V, Juan AH. Sculpting chromatin beyond the double helix: epigenetic control of skeletal myogenesis. In: Pavlath GK, editor. Myogenesis. United States: Elsevier Inc; 2011. p. 57–83.

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