Myostatin and activin blockade by engineered follistatin results in hypertrophy and improves dystrophic pathology in mdx mouse more than myostatin blockade alone-Reference-Cited by-同舟云学术

Myostatin and activin blockade by engineered follistatin results in hypertrophy and improves dystrophic pathology in mdx mouse more than myostatin blockade alone

Published:2018-10-27 Issue:1 Volume:8 Page:
ISSN:2044-5040
Container-title:Skeletal Muscle
language:en
Short-container-title:Skeletal Muscle

Author:

Iskenderian Andrea,Liu Nan,Deng Qingwei,Huang Yan,Shen Chuan,Palmieri Kathleen,Crooker Robert,Lundberg Dianna,Kastrapeli Niksa,Pescatore Brian,Romashko Alla,Dumas John,Comeau Robert,Norton Angela,Pan Jing,Rong Haojing,Derakhchan Katayoun,Ehmann David E.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Orthopedics and Sports Medicine

Link

http://link.springer.com/content/pdf/10.1186/s13395-018-0180-z.pdf

Reference74 articles.

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2. Guiraud S, Aartsma-Rus A, Vieira NM, Davies KE, van Ommen GJ, Kunkel LM. The pathogenesis and therapy of muscular dystrophies. Annu Rev Genom Hum G. 2015;16:281–308.

3. Ruegg UT. Pharmacological prospects in the treatment of Duchenne muscular dystrophy. Curr Opin Neurol. 2013;26(5):577–84.

4. Spinazzola JM, Kunkel LM. Pharmacological therapeutics targeting the secondary defects and downstream pathology of Duchenne muscular dystrophy. Expert Opin Orphan Drugs. 2016;4(11):1179–94.

5. Amthor H, Hoogaars WM. Interference with Myostatin/ActRIIB signaling as a therapeutic strategy for Duchenne muscular dystrophy. Curr Gene Ther. 2012;12(3):245–59.

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