Deletion of the α subunit of the heterotrimeric Go protein impairs cerebellar cortical development in mice-Reference-Cited by-同舟云学术

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Deletion of the α subunit of the heterotrimeric Go protein impairs cerebellar cortical development in mice

Published:2019-06-20 Issue:1 Volume:12 Page:
ISSN:1756-6606
Container-title:Molecular Brain
language:en
Short-container-title:Mol Brain

Author:

Cha Hye Lim,Choi Jung-Mi,Oh Huy-Hyen,Bashyal Narayan,Kim Sung-Soo,Birnbaumer Lutz,Suh-Kim Haeyoung^ORCID

Funder

National Research Foundation of Korea

National Institute of Environmental Health Sciences

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13041-019-0477-9.pdf

Reference59 articles.

1. Simon MI, Strathmann MP, Gautam N. Diversity of G proteins in signal transduction. Science. 1991;252:802–8.

2. Neer EJ. Heterotrimeric G proteins: organizers of transmembrane signals. Cell. 1995;80:249–57.

3. Birnbaumer L. Expansion of signal transduction by G proteins. The second 15 years or so: from 3 to 16 alpha subunits plus betagamma dimers. Biochim Biophys Acta. 2007;1768:772–93.

4. Rudolph U, Finegold MJ, Rich SS, Harriman GR, Srinivasan Y, Brabet P, et al. Gi2 alpha protein deficiency: a model of inflammatory bowel disease. J Clin Immunol. 1995;15:101S–5S.

5. Devanathan V, Hagedorn I, Kohler D, Pexa K, Cherpokova D, Kraft P, et al. Platelet Gi protein Galphai2 is an essential mediator of thrombo-inflammatory organ damage in mice. Proc Natl Acad Sci U S A. 2015;112:6491–6.

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