O-GlcNAc regulation of autophagy and α-synuclein homeostasis; implications for Parkinson’s disease-Reference-Cited by-同舟云学术

O-GlcNAc regulation of autophagy and α-synuclein homeostasis; implications for Parkinson’s disease

Published:2017-07-19 Issue:1 Volume:10 Page:
ISSN:1756-6606
Container-title:Molecular Brain
language:en
Short-container-title:Mol Brain

Author:

Wani Willayat Y.,Ouyang Xiaosen,Benavides Gloria A.,Redmann Matthew,Cofield Stacey S.,Shacka John J.,Chatham John C.,Darley-Usmar Victor,Zhang Jianhua^ORCID

Funder

MJFF

UAB AMC21

NIH

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13041-017-0311-1.pdf

Reference68 articles.

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3. Hara T, Nakamura K, Matsui M, Yamamoto A, Nakahara Y, Suzuki-Migishima R, Yokoyama M, Mishima K, Saito I, Okano H, et al. Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice. Nature. 2006;441:885–9.

4. Komatsu M, Waguri S, Chiba T, Murata S, Iwata JI, Tanida I, Ueno T, Koike M, Uchiyama Y, Kominami E, et al. Loss of autophagy in the central nervous system causes neurodegeneration in mice. Nature. 2006;441:880–4.

5. Nixon RA. The role of autophagy in neurodegenerative disease. Nat Med. 2013;19:983–97.

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