Estradiol dominance induces hemodilution and mild hematological alterations in mifepristone-treated rats
Author:
Affiliation:
1. Safety Research Department, R&D, Kissei Pharmaceuticals Co., Ltd.
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/47/7/47_301/_pdf
Reference34 articles.
1. Agarwai, M.K. (1996): The antiglucocorticoid action of mifepristone. Pharmacol. Ther., 70, 183-213.
2. Arbo, M.D., Schmitt, G.C., Limberger, M.F., Charão, M.F., Moro, A.M., Ribeiro, G.L., Dallegrave, E., Garcia, S.C., Leal, M.B. and Limberger, R.P. (2009): Subchronic toxicity of Citrus aurantium L. (Rutaceae) extract and p-synephrine in mice. Regul. Toxicol. Pharmacol., 54, 114-117.
3. Azushima, K., Morisawa, N., Tamura, K. and Nishiyama, A. (2020): Recent research advances in renin-angiotensin-aldosterone system receptors. Curr. Hypertens. Rep., 22, 22.
4. Clapham, J.C. and Turner, N.C. (1997): Effects of the glucocorticoid II receptor antagonist mifepristone on hypertension in the obese Zucker rat. J. Pharmacol. Exp. Ther., 282, 1503-1508.
5. Crofton, J.T. and Share, L. (1990): Sexual dimorphism in vasopressin and cardiovascular response to hemorrhage in the rat. Circ. Res., 66, 1345-1353.
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