Dose-dependent acceleration in the delayed effects of neonatal oral exposure to low-dose 17α-ethynylestradiol on reproductive functions in female Sprague-Dawley rats
Author:
Affiliation:
1. Laboratory of Comparative Toxicology, School of Veterinary Medicine
2. Laboratory of Veterinary Pathology, Azabu University
3. Research Institute of Biosciences, Azabu University
4. Division of Pathology, National Institute of Health Sciences
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/40/6/40_727/_pdf
Reference44 articles.
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2. Ayyanan, A., Laribi, O., Schuepbach-Mallepell, S., Schrick, C., Gutierrez, M., Tanos, T., Lefebvre, G., Rougemont, J., Yalcin-Ozuysal, O. and Brisken, C. (2011): Perinatal exposure to bisphenol A increases adult mammary gland progesterone response and cell number. Mol. Endocrinol., 25, 1915-1923.
3. Barkhem, T., Carlsson, B., Nilsson, Y., Enmark, E., Gustafsson, J. and Nilsson, S. (1998): Differential response of estrogen receptor α and estrogen receptor β to partial estrogen agonists/antagonists. Mol. Pharmacol., 54, 105-112.
4. Barraclough, C.A. (1961): Production of anovulatory, sterile rats by single injections of testosterone propionate. Endocrinology, 68, 62-67.
5. Betancourt, A.M., Eltoum, I.A., Desmond, R.A., Russo, J. and Lamartiniere, C.A. (2010): In utero exposure to bisphenol A shifts the window of susceptibility for mammary carcinogenesis in the rat. Environ. Health Perspect., 118, 1614-1619.
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