Arsenite suppresses NO production evoked by lipopolysaccharide and poly(I:C) via the suppression of interferon-β expression in RAW264.7 cells
Author:
Affiliation:
1. Laboratory of Molecular Nutrition and Toxicology, Faculty of Pharmaceutical Sciences, Tokushima Bunri University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/44/2/44_83/_pdf
Reference41 articles.
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2. Akaike, T., Suga, M. and Maeda, H. (1998): Free radicals in viral pathogenesis: molecular mechanisms involving superoxide and NO. Proc. Soc. Exp. Biol. Med., 217, 64-73.
3. Akaike, T., Weihe, E., Schaefer, M., Fu, Z.F., Zheng, Y.M., Vogel, W., Schmidt, H., Koprowski, H. and Dietzschold, B. (1995): Effect of neurotropic virus infection on neuronal and inducible nitric oxide synthase activity in rat brain. J. Neurovirol., 1, 118-125.
4. Banerjee, N., Banerjee, S., Sen, R., Bandyopadhyay, A., Sarma, N., Majumder, P., Das, J.K., Chatterjee, M., Kabir, S.N. and Giri, A.K. (2009): Chronic arsenic exposure impairs macrophage functions in the exposed individuals. J. Clin. Immunol., 29, 582-594.
5. Barchowsky, A., Dudek, E.J., Treadwell, M.D. and Wetterhahn, K.E. (1996): Arsenic induces oxidant stress and NF-kappa B activation in cultured aortic endothelial cells. Free Radic. Biol. Med., 21, 783-790.
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