Tributyltin activates the Keap1–Nrf2 pathway via a macroautophagy-independent reduction in Keap1
Author:
Affiliation:
1. Graduate School of Biomedical and Health Sciences, Hiroshima University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/48/3/48_161/_pdf
Reference45 articles.
1. Abiko, Y., Aoki, H. and Kumagai, Y. (2021): Effect of combined exposure to environmental aliphatic electrophiles from plants on Keap1/Nrf2 activation and cytotoxicity in HepG2 cells: A model of an electrophile exposome. Toxicol. Appl. Pharmacol., 413, 115392.
2. Bae, S.H., Sung, S.H., Oh, S.Y., Lim, J.M., Lee, S.K., Park, Y.N., Lee, H.E., Kang, D. and Rhee, S.G. (2013): Sestrins activate Nrf2 by promoting p62-dependent autophagic degradation of Keap1 and prevent oxidative liver damage. Cell Metab., 17, 73-84.
3. Baird, L. and Dinkova-Kostova, A.T. (2011): The cytoprotective role of the Keap1-Nrf2 pathway. Arch. Toxicol., 85, 241-272.
4. Baird, L. and Yamamoto, M. (2020): The Molecular Mechanisms Regulating the KEAP1-NRF2 Pathway. Mol. Cell. Biol., 40, e00099-e20.
5. Canning, P., Sorrell, F.J. and Bullock, A.N. (2015): Structural basis of Keap1 interactions with Nrf2. Free Radic. Biol. Med., 88, 101-107.
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