Inhibition of VDAC1 prevents oxidative stress and apoptosis induced by bisphenol A in spermatogonia via AMPK/mTOR signaling pathway
Author:
Affiliation:
1. Department of Obstetrics and Gynecology, the First Affiliated Hospital of The Fourth Military Medical University, China
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/48/3/48_109/_pdf
Reference61 articles.
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2. Al-Hiyasat, A.S., Darmani, H. and Elbetieha, A.M. (2002): Effects of bisphenol A on adult male mouse fertility. Eur. J. Oral Sci., 110, 163-167.
3. Amir, A., Yanwirasti, Asmarinah and Oenzil, F. (2016): Alteration Expression of Bax, Bcl-2 and VDAC1 Genes in Oligozoospermic and Fertile Subjects. Pakistan journal of biological sciences: PJBS, 19, 71-76.
4. Castellini, C., Totaro, M., Parisi, A., D’Andrea, S., Lucente, L., Cordeschi, G., Francavilla, S., Francavilla, F. and Barbonetti, A. (2020): Bisphenol A and Male Fertility: myths and Realities. Front. Endocrinol. (Lausanne), 11, 353.
5. Chen, H., Gao, W., Yang, Y., Guo, S., Wang, H., Wang, W., Zhang, S., Zhou, Q., Xu, H., Yao, J., Tian, Z., Li, B., Cao, W., Zhang, Z. and Tian, Y. (2014): Inhibition of VDAC1 prevents Ca2+-mediated oxidative stress and apoptosis induced by 5-aminolevulinic acid mediated sonodynamic therapy in THP-1 macrophages. Apoptosis: an international journal on programmed cell death, 19, 1712-1726.
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