Social behavior, neuroimmune markers and glutamic acid decarboxylase levels in a rat model of valproic acid-induced autism
Author:
Affiliation:
1. National Institute for Environmental Studies
2. University of Medicine 1, Myanmar
3. Graduate School of Medical Sciences, Kitasato University, Japan
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/43/11/43_631/_pdf
Reference58 articles.
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2. Baltz, T., de Lima, A.D. and Voigt, T. (2010): Contribution of GABAergic interneurons to the development of spontaneous activity patterns in cultured neocortical networks. Front. Cell. Neurosci., 4, 15.
3. Banerjee, A., García-Oscos, F., Roychowdhury, S., Galindo, L.C., Hall, S., Kilgard, M.P. and Atzori, M. (2013): Impairment of cortical GABAergic synaptic transmission in an environmental rat model of autism. Int. J. Neuropsychopharmacol., 16, 1309-1318.
4. Barrett, C.E., Hennessey, T.M., Gordon, K.M., Ryan, S.J., McNair, M.L., Ressler, K.J. and Rainnie, D.G. (2017): Developmental disruption of amygdala transcriptome and socioemotional behavior in rats exposed to valproic acid prenatally. Mol. Autism, 8, 42.
5. Benes, F.M. and Berretta, S. (2001): GABAergic interneurons: implications for understanding schizophrenia and bipolar disorder. Neuropsychopharmacology, 25, 1-27.
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