Relationship between low midazolam metabolism by cytochrome P450 3A in mice and the high incidence of birth defects
Author:
Affiliation:
1. Department of Clinical Pharmacokinetics, Hoshi University
2. Department of Functional Molecule Kinetics Hoshi University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/43/1/43_65/_pdf
Reference23 articles.
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2. Dai, D., Bai, R., Hodgson, E. and Rose, R.L. (2001): Cloning, sequencing, heterologous expression, and characterization of murine cytochrome P450 3a25*(Cyp3a25), a testosterone 6beta-hydroxylase. J. Biochem. Mol. Toxicol., 15, 90-99.
3. Eberts, F.S. Jr., Philopoulos, Y., Reineke, L.M. and Vliek, R.W. (1981): Triazolam disposition. Clin. Pharmacol. Ther., 29, 81-93.
4. Handel, J., Ziegler, G., Gemeinhardt, A., Stuber, H., Fischer, C. and Klotz, U. (1988): Lack of effect of nitrendipine on the pharmacokinetics and pharmacodynamics of midazolam during steady state. Br. J. Clin. Pharmacol., 25, 243-250.
5. Hart, S.N., Cui, Y., Klaassen, C.D. and Zhong, X.B. (2009): Three patterns of cytochrome P450 gene expression during liver maturation in mice. Drug Metab. Dispos., 37, 116-121.
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