Effects of hyperprolactinemia on toxicological parameters and proliferation of islet cells in male rats
Author:
Affiliation:
1. Environmental Health Science Laboratory, Sumitomo Chemical Company, Ltd.
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/34/2/34_2_151/_pdf
Reference33 articles.
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2. Baptista, T., de Baptista, E.A., Kin, N.M.K.Ng.Y., Beaulieu, S., Walker, D., Joober, R., Lalonde, J. and Richard, D. (2002): Comparative effects of the antipsychotics sulpiride or risperidone in rats: I: Bodyweight, food intake, body composition, hormones and glucose tolerance. Brain Res., 957, 144-151.
3. Boot, L.M., Mühlbock, O., Röpcke, G. and Van Ebbenhorst Tenbergen W. (1962): Further investigations on induction of mammary cancer in mice by isografts of hypophyseal tissue. Cancer Res., 22, 713-727.
4. Brelje, T.C., Parsons, J.A. and Sorenson, R.L. (1994): Regulation of islet β-cell proliferation by prolactin in rat islets. Diabetes, 43, 263-273.
5. Brelje, T.C., Scharp, D.W., Lacy, P.E., Ogren, L., Talamantes, F., Robertson, M., Friesen, H.G. and Sorenson, R.L. (1993): Effect of homologous placental lactogens, prolactins, and growth hormones on islet B-cell division and insulin secretion in rat, mouse, and human islets: Implication for placental lactogen regulation of islet function during pregnancy. Endocrinolgy, 132, 879-887.
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