NF-κB activation via MyD88-dependent Toll-like receptor signaling is inhibited by trichothecene mycotoxin deoxynivalenol-Reference-Cited by-同舟云学术

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NF-κB activation via MyD88-dependent Toll-like receptor signaling is inhibited by trichothecene mycotoxin deoxynivalenol

Published:2016 Issue:2 Volume:41 Page:273-279
ISSN:0388-1350
Container-title:The Journal of Toxicological Sciences
language:en
Short-container-title:J. Toxicol. Sci.

Author:

Sugiyama Kei-ichi¹,Muroi Masashi²,Kinoshita Mawo¹³,Hamada Osamu¹³,Minai Yuji³,Sugita-Konishi Yoshiko⁴,Kamata Yoichi⁵,Tanamoto Ken-ichi²

Affiliation:

1. Division of Genetics and Mutagenesis, National Institute of Health Sciences

2. Research Institute of Pharmaceutical Sciences, Musashino University

3. Department of Applied Biological Chemistry, Faculty of Agriculture, Tamagawa University

4. Department of Food and Life Sciences, Azabu University

5. Department of Veterinary Science, Faculty of Agriculture, Iwate University

Publisher

Japanese Society of Toxicology

Subject

Toxicology

Link

https://www.jstage.jst.go.jp/article/jts/41/2/41_273/_pdf

Reference42 articles.

1. Akira, S., Uematsu, S. and Takeuchi, O. (2006): Pathogen recognition and innate immunity. Cell, 124, 783-801.

2. Bennett, J.W. and Klich, M. (2003): Mycotoxins. Clin. Microbiol. Rev., 16, 497-516.

3. Beutler, B.A. (2009): TLRs and innate immunity. Blood, 113, 1399-1407.

4. Boockvar, K.S., Granger, D.L., Poston, R.M., Maybodi, M., Washington, M.K., Hibbs, J.B.Jr. and Kurlander, R.L. (1994): Nitric oxide produced during murine listeriosis is protective. Infect. Immun., 62, 1089-1100.

5. Chen, Z., Hagler, J., Palombella, V.J., Melandri, F., Scherer, D., Ballard, D. and Maniatis, T. (1995): Signal-induced site-specific phosphorylation targets IκBα to the ubiquitin-proteasome pathway. Genes Dev., 9, 1586-1597.

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